Metabolic Responses of the Rooster After Exogenous Thyroid Hormones
نویسندگان
چکیده
منابع مشابه
Thyroid hormones and the metabolic syndrome.
BACKGROUND Clustering of various metabolic parameters including abdominal obesity, hyperglycaemia, low high-density lipoprotein cholesterol, elevated triglycerides and hypertension have been used worldwide as metabolic syndrome to predict cardiometabolic risk. Thyroid dysfunction impacts on various levels of these components. OBJECTIVES The purpose of the present review is to summarize availa...
متن کاملInteractions of metabolic syndrome and thyroid hormones
The prevalence of metabolic syndrome worldwide is high and rising (Alexander Diabetes 52: 1210, 2003; Cameron Endocrinol Metab Clin North Am 33: 351, 2004). According to all definitions visceral obesity, increased triglycerides, decreased HDL cholesterol, impaired glucose tolerance and increased blood pressure are part of the definition. Thyroid hormones impact on all these factors. In a large ...
متن کاملThyroid hormones – obesity and metabolic syndrome
Recently there has been increased interest in the association between thyroid function and obesity. Based on the notion that triiodothyronine (T3) controls metabolic and energy homeostasis and influences body weight, thermogenesis, lipolysis and metabolism of cholesterol, and that thyroid-stimulating hormone (TSH) via receptors in fat tissue, induces differentiation of preadipocytes in to adipo...
متن کاملAssociation between thyroid hormones, insulin resistance, and metabolic syndrome.
OBJECTIVE To determine the association between thyroid hormones, insulin resistance, and metabolic syndrome in euthyroid women. METHODS Forty-five women with no past medical history were studied in this cross-sectional study at the Department of Endocrinology, Medwin Hospitals, Hyderabad, India, from August 2008 to September 2008. The body fat was estimated using bio-impedance method, and fas...
متن کاملThyroid hormones inhibit TGF-β signaling and attenuate fibrotic responses.
TGF-β, the most potent profibrogenic factor, acts by activating SMAD (mothers against decapentaplegic) transcription factors, which bind to SMAD-binding elements in target genes. Here, we show that the thyroid hormone triiodothyronine (T3), through binding to its nuclear receptors (TRs), is able to antagonize transcriptional activation by TGF-β/SMAD. This antagonism involves reduced phosphoryla...
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ژورنال
عنوان ژورنال: Poultry Science
سال: 1982
ISSN: 0032-5791
DOI: 10.3382/ps.0611748